By Dr. Reena R. Kumar
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Extra resources for Journal of Orofacial & Health Sciences
BP inhibits bone resorption and causes increased bone density which may adversely affect orthodontic tooth movement, impair bone healing and may even cause osteonecrosis of the jaw. This article presents a briefoverview of the applied aspects ofpharmacology, pharmacokinetics of the drug and the guidelines to plan and progress orthodontic treatment in patients on bisphosphonate therapy Key Words: Bisphosphonate, Orthodontic Treatment appropriate INTRODUCTION The current orthodontic patient clientele has undergone a transformation with respect to growing numbers of adults seeking orthodontic aesthetic and functional attention.
Intra-oral periapical radiographs the lesions1,2. their spatial relationship with anatomic are still most commonly exposed during endodontic landmarks3 , it has been proposed that the amount of procedures, providing useful information for the presence bone that has to be resorbed before a lesion becomes and location of periradicular lesions, root canal anatomy clearly visible is quite extensive 3 . ' Radiographs are limited to visualization of hard tissues (Fig 1) and; therefore, they cannot guide the clinician towards a diagnosis of the soft tissue characteristics of the lesions 4.
The mevalonate pathway. 2,9 Orthodontist should bear in mind that treatment of any severe medical condition or prevention of fractures has priority over orthodontic treatment, hence discontinuation of the drug by the orthodontist is not an available option. The prescribing physician may decide to change or discontinue bisphosphonates after careful riskbenefit analysis for each patient. bone surface and in the blood. e. orthodontic patients into high and low risk groups. Patients on long tooth movements should be completed in 6-9 months duration of drug, especially nitrogenous group, IV route of period.