Dihydrotestosterone - A Medical Dictionary, Bibliography, by Icon Health Publications

By Icon Health Publications

In March 2001, the nationwide Institutes of healthiness issued the subsequent caution: "The variety of sites delivering health-related assets grows each day. Many websites supply beneficial info, whereas others can have details that's unreliable or misleading." additionally, as a result of swift raise in Internet-based info, many hours will be wasted looking out, deciding upon, and printing.This publication used to be created for doctors, scholars, and contributors of most of the people who are looking to behavior clinical learn utilizing the main complex instruments to be had and spending the smallest amount of time doing so.

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Additional resources for Dihydrotestosterone - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References

Sample text

Determination if treatment with estrogen reduces the severity of and/or accelerates recovery from incontinence symptoms and nerve damage after vaginal distension, and SA4. Determination if treatment with dihydrotestosterone reduces the severity of and/or accelerates recovery from incontinence symptoms and nerve damage after vaginal distension. These Specific Aims will be tested in an established animal model of 34 Dihydrotestosterone vaginal distension by urodynamic testing, histological evidence, and 3u tubulin mRNA levels in pudendal motoneurons determined using in situ hybridization.

Growth hormone (GH) increases alcohol dehydrogenase by increasing its synthesis at the level of transcription, while dihydrotestosterone decreases alcohol dehydrogenase by increasing its rate of degradation. It will now be determined whether the effect of GH in enhancing transcription (STAT) proteins. The pathway for degradation of alcohol dehydrogenase will be defined by determining if specific inhibitors of various pathways prevent the effect of dihydrotestosterone in increasing degradation of the enzyme.

For example, toward this goal, we have previously developed a selective topical liposome hair follicle targeting technology fo r genes and other large and small molecules. The present application will focus on our recent observation that the 5-alpha- reductase inhibitor N, N-diethyl-4-methyl-3-oxo-4-aza-5alpha- androstane17beta-carboxamide (4-MA) incorporated into liposomes selectively induces apoptosis and inhibits growth of the dihydrotestosterone (DHT)-dependent hamster flank organ sebaceous gland.

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